VX, resembling engine oil in consistency, originated from ICI's research on novel insecticides during the early 1950s. However, its extreme toxicity precluded its agricultural use. VX is lethal by disrupting the transmission of nerve signals between cells, which relies on the presence of acetylcholine. Once acetylcholine has transmitted its signal, it must be broken down to prevent continuous signaling by an enzyme catalyst known as acetylcholinesterase. Nerve agents like VX inhibit this enzyme, resulting in uncontrolled muscle contractions and ultimately leading to death due to asphyxiation.